1.What are the clinical features of a ruptured sinus of Valsalva?

2.Does dobutamine (dose range 2.5-10 μg/kg/min) cause significant

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3.Are beta-blockers indicated in heart failure; if yes, in all cases or in

selected cases?

4.Why does long-term treatment by digitalis cause gynaecomastia?

5.Why do toxic doses of digoxin (which cause a decrease in excitability of

cardiac tissue) cause arrhythmias, whereas therapeutic levels of digoxin

(which cause increase in excitability) cause no arrhythmias?

6.Hypokalaemia is one of the complications of digitalis. In treatment

we give K. How does the hyperkalaemia enhance the digitalis


7.Why should we measure serum potassium before we start digoxin?

8.The intravenous administration of loop diuretics such as furosemide

relieves pulmonary oedema rapidly. Is this due to arteriolar

vasodilatation reducing afterload which is an action that is independent

of its diuretic effect? I ask this because I have been told that this drug has

no effect on arterioles (except on efferent arterioles of the kidney), but

rather a venodilatatory effect. What could be the cause of such different


9.There have been a number of recent publications concerning aspirin

versus warfarin trials. Would you recommend prescribing warfarin for

secondary prevention of coronary heart disease in patients suffering from

their first myocardial infarction (MI)?

10 Is it true that in future the ApoA : ApoB ratio will be used to predict

the risk of coronary artery disease more efficiently than low-density

lipoprotein (LDL)?

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